Research
Brain Trauma: plasticity, blood flow & metabolism, neurogenesis, PET, MRI
Appointments
- Associate Professor, Neurosurgery
- Member, Brain Research Institute
- Cell & Developmental Biology GPB Home Area
- Neuroscience GPB Home Area
Biography
Dr. Neil Harris has worked within the fields of hydrocephalus, stroke and traumatic brain injury. The major theme of his current research at UCLA is understanding the potential for plasticity and function after trauma utilizing both light microscopy cell biology techniques as well as in vivo neuroimaging methodologies.
Dr. Harris holds an RO1 award to examine axonal plasticity and functional recovery after traumatic brain injury.
Publications
- Golovachev, N, Siebold, L, Sutton, RL, Ghavim, S, Harris, NG, Bartnik-Olson, B et al.. Metabolic-driven analytics of traumatic brain injury and neuroprotection by ethyl pyruvate. J Neuroinflammation. 2024;21 (1):294. doi: 10.1186/s12974-024-03280-8. PubMed PMID:39538295 PubMed Central PMC11562096.
- Santana-Gomez, C, Smith, G, Mousavi, A, Shamas, M, Harris, NG, Staba, R et al.. The Surgical Method of Craniectomy Differentially Affects Acute Seizures, Brain Deformation, and Behavior in a Traumatic Brain Injury Animal Model. Neurotrauma Rep. 2024;5 (1):969-981. doi: 10.1089/neur.2024.0064. PubMed PMID:39440152 PubMed Central PMC11491586.
- Kim, Y, Hrncir, H, Meyer, CE, Tabbaa, M, Moats, RA, Levitt, P et al.. Mouse Brain Extractor: Brain segmentation of mouse MRI using global positional encoding and SwinUNETR. bioRxiv. 2024; :. doi: 10.1101/2024.09.03.611106. PubMed PMID:39282435 PubMed Central PMC11398355.
- Paydar, A, Khorasani, L, Harris, NG. Constraint Induced Movement Therapy Confers only a Transient Behavioral Benefit but Enduring Functional Circuit-Level Changes after Experimental TBI. bioRxiv. 2024; :. doi: 10.1101/2024.08.02.606449. PubMed PMID:39149371 PubMed Central PMC11326145.
- Le Belle, JE, Condro, M, Cepeda, C, Oikonomou, KD, Tessema, K, Dudley, L et al.. Acute rapamycin treatment reveals novel mechanisms of behavioral, physiological, and functional dysfunction in a maternal inflammation mouse model of autism and sensory over-responsivity. bioRxiv. 2024; :. doi: 10.1101/2024.07.08.602602. PubMed PMID:39026891 PubMed Central PMC11257517.